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Publications by Maged Harraz

Iron-Mediated H2O2Production as a Mechanism for Cell Type-Specific Inhibition of Tumor Necrosis Factor Α-Induced but Not Interleukin-1β-Induced IκB Kinase Complex/Nuclear Factor-κB Activation

Journal of Biological Chemistry

English

Related publications

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

Journal of Biological Chemistry

English

NF-κB; IL-1β; Tumor Necrosis Factor-Α (TNF-α)

Science-Business eXchange

2009

English

Glutathione Depletion Down-Regulates Tumor Necrosis Factor Α-Induced NF-κB Activity via IκB Kinase-Dependent and -Independent Mechanisms

Journal of Biological Chemistry

English

Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis

Journal of Biological Chemistry

English

Tumor Necrosis Factor-Α-Induced Cell Killing and Activation of Transcription Factor NF-κB Are Uncoupled in L929 Cells

Journal of Biological Chemistry

English

A Promoter Recruitment Mechanism for Tumor Necrosis Factor-Α-Induced Interleukin-8 Transcription in Type II Pulmonary Epithelial Cells

Journal of Biological Chemistry

English

Tumor Necrosis Factor-Α Induces Transcriptional Activation of Nuclear Factor-κB in Insulin-Producing Β-Cells

Experimental and Therapeutic Medicine

English

Acid Ceramidase but Not Acid Sphingomyelinase Is Required for Tumor Necrosis Factor-Α-Induced PGE2Production

Journal of Biological Chemistry

English

Inducible IκB Kinase/IκB Kinase Ε Expression Is Induced by CK2 and Promotes Aberrant Nuclear Factor-κB Activation in Breast Cancer Cells

Cancer Research

Oncology

2005

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Maged Harraz

Iron-Mediated H2O2Production as a Mechanism for Cell Type-Specific Inhibition of Tumor Necrosis Factor Α-Induced but Not Interleukin-1β-Induced IκB Kinase Complex/Nuclear Factor-κB Activation

Related publications

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

NF-κB; IL-1β; Tumor Necrosis Factor-Α (TNF-α)

Glutathione Depletion Down-Regulates Tumor Necrosis Factor Α-Induced NF-κB Activity via IκB Kinase-Dependent and -Independent Mechanisms

Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis

Tumor Necrosis Factor-Α-Induced Cell Killing and Activation of Transcription Factor NF-κB Are Uncoupled in L929 Cells

A Promoter Recruitment Mechanism for Tumor Necrosis Factor-Α-Induced Interleukin-8 Transcription in Type II Pulmonary Epithelial Cells

Tumor Necrosis Factor-Α Induces Transcriptional Activation of Nuclear Factor-κB in Insulin-Producing Β-Cells

Acid Ceramidase but Not Acid Sphingomyelinase Is Required for Tumor Necrosis Factor-Α-Induced PGE2Production

Inducible IκB Kinase/IκB Kinase Ε Expression Is Induced by CK2 and Promotes Aberrant Nuclear Factor-κB Activation in Breast Cancer Cells