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Publications by Meike Mitsdoerffer
IL-21R Signaling Is Critical for Induction of Spontaneous Experimental Autoimmune Encephalomyelitis
Journal of Clinical Investigation
Medicine
Related publications
MAdCAM-1-Mediated Intestinal Lymphocyte Homing Is Critical for the Development of Active Experimental Autoimmune Encephalomyelitis
Frontiers in Immunology
Allergy
Immunology
IL-12p35-Deficient Mice Are Susceptible to Experimental Autoimmune Encephalomyelitis: Evidence for Redundancy in the IL-12 System in the Induction of Central Nervous System Autoimmune Demyelination
Journal of Immunology
Allergy
Immunology
Coordinated Induction of Extracellular Proteolysis Systems During Experimental Autoimmune Encephalomyelitis in Mice
American Journal of Pathology
Forensic Medicine
Pathology
IL-21 Receptor (IL-21R)
Science-Business eXchange
Mechanical Hypernociception in Experimental Autoimmune Encephalomyelitis
Arquivos de Neuro-Psiquiatria
Biological Psychiatry
Neurology
Cutting Edge: Macrophage Migration Inhibitory Factor Is Necessary for Progression of Experimental Autoimmune Encephalomyelitis
Journal of Immunology
Allergy
Immunology
Complement in Experimental Autoimmune Encephalomyelitis Revisited: C3 Is Required for Development of Maximal Disease
Molecular Immunology
Immunology
Molecular Biology
Effect of Targeted Disruption of STAT4 and STAT6 on the Induction of Experimental Autoimmune Encephalomyelitis
Journal of Clinical Investigation
Medicine
A Tale of Two STAT6 Knock Out Mice in the Induction of Experimental Autoimmune Encephalomyelitis
Journal of Neuroimmunology
Allergy
Immunology
Neurology
