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Publications by Nand K. Sah

Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis

Journal of Biological Chemistry

English

Related publications

Tumor Necrosis Factor-Regulated Biphasic Activation of NF-κB Is Required for Cytokine-Induced Loss of Skeletal Muscle Gene Products

Journal of Biological Chemistry

English

Functional P53 Is Required for Triptolide-Induced Apoptosis and AP-1 and Nuclear Factor-κB Activation in Gastric Cancer Cells

English

Lithium Sensitizes Tumor Cells in an NF-κB-independent Way to Caspase Activation and Apoptosis Induced by Tumor Necrosis Factor (TNF)

Journal of Biological Chemistry

English

Iron-Mediated H2O2Production as a Mechanism for Cell Type-Specific Inhibition of Tumor Necrosis Factor Α-Induced but Not Interleukin-1β-Induced IκB Kinase Complex/Nuclear Factor-κB Activation

Journal of Biological Chemistry

English

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

Journal of Biological Chemistry

English

Acid Ceramidase but Not Acid Sphingomyelinase Is Required for Tumor Necrosis Factor-Α-Induced PGE2Production

Journal of Biological Chemistry

English

NF-κB Mediates Tumor Necrosis Factor Α-Induced Expression of Optineurin, a Negative Regulator of NF-κB

PLoS ONE

Multidisciplinary

2009

English

Cross-Talk Between Receptor Tyrosine Kinase and Tumor Necrosis Factor-Α Signaling Networks Regulates Apoptosis but Not Proliferation

Molecular and Cellular Proteomics

English

Tumor Necrosis Factor-Α-Induced Cell Killing and Activation of Transcription Factor NF-κB Are Uncoupled in L929 Cells

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Nand K. Sah

Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis

Related publications

Tumor Necrosis Factor-Regulated Biphasic Activation of NF-κB Is Required for Cytokine-Induced Loss of Skeletal Muscle Gene Products

Functional P53 Is Required for Triptolide-Induced Apoptosis and AP-1 and Nuclear Factor-κB Activation in Gastric Cancer Cells

Lithium Sensitizes Tumor Cells in an NF-κB-independent Way to Caspase Activation and Apoptosis Induced by Tumor Necrosis Factor (TNF)

Iron-Mediated H2O2Production as a Mechanism for Cell Type-Specific Inhibition of Tumor Necrosis Factor Α-Induced but Not Interleukin-1β-Induced IκB Kinase Complex/Nuclear Factor-κB Activation

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

Acid Ceramidase but Not Acid Sphingomyelinase Is Required for Tumor Necrosis Factor-Α-Induced PGE2Production

NF-κB Mediates Tumor Necrosis Factor Α-Induced Expression of Optineurin, a Negative Regulator of NF-κB

Cross-Talk Between Receptor Tyrosine Kinase and Tumor Necrosis Factor-Α Signaling Networks Regulates Apoptosis but Not Proliferation

Tumor Necrosis Factor-Α-Induced Cell Killing and Activation of Transcription Factor NF-κB Are Uncoupled in L929 Cells