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Publications by Ute Wittig

Live-Cell Imaging of Endogenous Ras-GTP Illustrates Predominant Ras Activation at the Plasma Membrane

EMBO Reports

English

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The Structural Basis for the Transition From Ras-GTP to Ras-GDP

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The Deubiquitinating Enzyme USP17 Blocks N-Ras Membrane Trafficking and Activation but Leaves K-Ras Unaffected

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Galectin-1 Binds Oncogenic H-Ras to Mediate Ras Membrane Anchorage and Cell Transformation

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Colocalization of Ras and Ral on the Membrane Is Required for Ras-Dependent Ral Activation Through Ral GDP Dissociation Stimulator

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In Vitro Expression of NGN3 Identifies RAB3B as the Predominant Ras-Associated GTP-binding Protein 3 Family Member in Human Islets

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Galectin-1 Augments Ras Activation and Diverts Ras Signals to Raf-1 at the Expense of Phosphoinositide 3-Kinase

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Ras-GTP Dimers Activate the Mitogen-Activated Protein Kinase (MAPK) Pathway

Proceedings of the National Academy of Sciences of the United States of America

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Raf Activation by Ras and Promotion of Cellular Metastasis Require Phosphorylation of Prohibitin in the Raft Domain of the Plasma Membrane

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Differential Interaction of the Ras Family GTP-binding Proteins H-Ras, Rap1A, and R-Ras With the Putative Effector Molecules Raf Kinase and Ral-Guanine Nucleotide Exchange Factor

Journal of Biological Chemistry

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Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Ute Wittig

Live-Cell Imaging of Endogenous Ras-GTP Illustrates Predominant Ras Activation at the Plasma Membrane

Related publications

The Structural Basis for the Transition From Ras-GTP to Ras-GDP

The Deubiquitinating Enzyme USP17 Blocks N-Ras Membrane Trafficking and Activation but Leaves K-Ras Unaffected

Galectin-1 Binds Oncogenic H-Ras to Mediate Ras Membrane Anchorage and Cell Transformation

Colocalization of Ras and Ral on the Membrane Is Required for Ras-Dependent Ral Activation Through Ral GDP Dissociation Stimulator

In Vitro Expression of NGN3 Identifies RAB3B as the Predominant Ras-Associated GTP-binding Protein 3 Family Member in Human Islets

Galectin-1 Augments Ras Activation and Diverts Ras Signals to Raf-1 at the Expense of Phosphoinositide 3-Kinase

Ras-GTP Dimers Activate the Mitogen-Activated Protein Kinase (MAPK) Pathway

Raf Activation by Ras and Promotion of Cellular Metastasis Require Phosphorylation of Prohibitin in the Raft Domain of the Plasma Membrane

Differential Interaction of the Ras Family GTP-binding Proteins H-Ras, Rap1A, and R-Ras With the Putative Effector Molecules Raf Kinase and Ral-Guanine Nucleotide Exchange Factor