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Publications by Y. Tsujimoto

Proapoptotic BH3-only BCL-2 Family Members Induce Cytochrome C Release, but Not Mitochondrial Membrane Potential Loss, and Do Not Directly Modulate Voltage-Dependent Anion Channel Activity

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

2000

English

Localization of the Human Pim Oncogene (PIM) to a Region of Chromosome 6 Involved in Translocations in Acute Leukemias.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1986

English

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An Interaction Between BCL-xLand the Voltage-Dependent Anion Channel (VDAC) Promotes Mitochondrial Ca2+Uptake

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BCL-2 Family Members Do Not Inhibit Apoptosis by Binding the Caspase Activator Apaf-1

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1999

English

BCL-2 Prevents Mitochondrial Permeability Transition and Cytochrome C Release via Maintenance of Reduced Pyridine Nucleotides

Cell Death and Differentiation

Cell Biology

Molecular Biology

2000

English

Bfk: A Novel Weakly Proapoptotic Member of the BCL-2 Protein Family With a BH3 and a BH2 Region

Cell Death and Differentiation

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2003

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The BCL-2 Protein Family, BH3-mimetics and Cancer Therapy

Cell Death and Differentiation

Cell Biology

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2015

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Glycogen Synthase Kinase 3-Mediated Voltage-Dependent Anion Channel Phosphorylation Controls Outer Mitochondrial Membrane Permeability During Lipid Accumulation

English

Voltage-Dependent Membrane Properties Shape the Size but Not the Frequency Content of Spontaneous Voltage Fluctuations in Layer 2/3 Somatosensory Cortex

Journal of Neuroscience

Neuroscience

2019

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Not Only More but Better

Chemical and Engineering News

Chemical Engineering

1962

English

Experimental Characterization of the Binding Affinities Between Proapoptotic BH3 Peptides and Antiapoptotic BCL-2 Proteins

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Y. Tsujimoto

Proapoptotic BH3-only BCL-2 Family Members Induce Cytochrome C Release, but Not Mitochondrial Membrane Potential Loss, and Do Not Directly Modulate Voltage-Dependent Anion Channel Activity

Localization of the Human Pim Oncogene (PIM) to a Region of Chromosome 6 Involved in Translocations in Acute Leukemias.

Related publications

An Interaction Between BCL-xLand the Voltage-Dependent Anion Channel (VDAC) Promotes Mitochondrial Ca2+Uptake

BCL-2 Family Members Do Not Inhibit Apoptosis by Binding the Caspase Activator Apaf-1

BCL-2 Prevents Mitochondrial Permeability Transition and Cytochrome C Release via Maintenance of Reduced Pyridine Nucleotides

Bfk: A Novel Weakly Proapoptotic Member of the BCL-2 Protein Family With a BH3 and a BH2 Region

The BCL-2 Protein Family, BH3-mimetics and Cancer Therapy

Glycogen Synthase Kinase 3-Mediated Voltage-Dependent Anion Channel Phosphorylation Controls Outer Mitochondrial Membrane Permeability During Lipid Accumulation

Voltage-Dependent Membrane Properties Shape the Size but Not the Frequency Content of Spontaneous Voltage Fluctuations in Layer 2/3 Somatosensory Cortex

Not Only More but Better

Experimental Characterization of the Binding Affinities Between Proapoptotic BH3 Peptides and Antiapoptotic BCL-2 Proteins