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Publications by Yicheng Feng
Mycobacterium Tuberculosis Dihydrofolate Reductase Is Not a Target Relevant to the Antitubercular Activity of Isoniazid
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
Related publications
Acetylation of Isoniazid - A Novel Mechanism of Isoniazid Resistance in Mycobacterium Tuberculosis
The Isoniazid-Nad Adduct Is a Slow, Tight-Binding Inhibitor of InhA, the Mycobacterium Tuberculosis Enoyl Reductase: Adduct Affinity and Drug Resistance
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Kinetic and Chemical Mechanism of the Dihydrofolate Reductase fromMycobacterium Tuberculosis
Biochemistry
Biochemistry
Action Mechanism of Antitubercular Isoniazid
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Proteome-Wide Profiling of Isoniazid Targets in Mycobacterium Tuberculosis
The Structure of the Catalytic Domain of the ATP Synthase From Mycobacterium Smegmatis Is a Target for Developing Antitubercular Drugs
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Observed Crowding Effects on Mycobacterium Tuberculosis 2-Trans-Enoyl-Acp (CoA) Reductase Enzyme Activity Are Not Due to Excluded Volume Only
Scientific Reports
Multidisciplinary
Isoniazid Induces Expression of the Antigen 85 Complex in Mycobacterium Tuberculosis.
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
Isoniazid-Induced Transient High-Level Resistance in Mycobacterium Tuberculosis
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
