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Publications by J Vlach

Cooperation Between Herpes Simplex Virus Type 1-Encoded ICP0 and Tat to Support Transcription of Human Immunodeficiency Virus Type 1 Long Terminal Repeat in Vivo Can Occur in the Absence of the TAR Binding Site.

Journal of Virology

English

Isolation of Pure Anhydrotetracycline Oxygenase fromStreptomyces Aureofaciens

English

Related publications

Interactions of Thyroid Hormone Receptor With the Human Immunodeficiency Virus Type 1 (HIV-1) Long Terminal Repeat and the HIV-1 Tat Transactivator.

English

Repression in Vitro, by Human Adenovirus E1A Protein Domains, of Basal or Tat-Activated Transcription of the Human Immunodeficiency Virus Type 1 Long Terminal Repeat.

English

Molecular and Functional Interactions of Transcription Factor USF With the Long Terminal Repeat of Human Immunodeficiency Virus Type 1.

English

The TAR Hairpin of Human Immunodeficiency Virus Type 1 Can Be Deleted When Not Required for Tat-Mediated Activation of Transcription

English

Stimulation of Tat-Associated Kinase-Independent Transcriptional Elongation From the Human Immunodeficiency Virus Type-1 Long Terminal Repeat by a Cellular Enhancer

English

Transcription Factor Binding Sites Downstream of the Human Immunodeficiency Virus Type 1 Transcription Start Site Are Important for Virus Infectivity.

English

Antiviral Activities of Glycyrrhizin and Its Modified Compounds Against Human Immunodeficiency Virus Type 1(hiv-1) and Herpes Simplex Virus Type 1(hsv-1) in Vitro.

Chemical and Pharmaceutical Bulletin

English

Involvement of Long Terminal Repeat U3 Sequences Overlapping the Transcription Control Region in Human Immunodeficiency Virus Type 1 mRNA 3' End Formation.

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1991

English

Ribonucleotide Reductase of Herpes Simplex Virus Type 2 Resembles That of Herpes Simplex Virus Type 1.

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by J Vlach

Cooperation Between Herpes Simplex Virus Type 1-Encoded ICP0 and Tat to Support Transcription of Human Immunodeficiency Virus Type 1 Long Terminal Repeat in Vivo Can Occur in the Absence of the TAR Binding Site.

Isolation of Pure Anhydrotetracycline Oxygenase fromStreptomyces Aureofaciens

Related publications

Interactions of Thyroid Hormone Receptor With the Human Immunodeficiency Virus Type 1 (HIV-1) Long Terminal Repeat and the HIV-1 Tat Transactivator.

Repression in Vitro, by Human Adenovirus E1A Protein Domains, of Basal or Tat-Activated Transcription of the Human Immunodeficiency Virus Type 1 Long Terminal Repeat.

Molecular and Functional Interactions of Transcription Factor USF With the Long Terminal Repeat of Human Immunodeficiency Virus Type 1.

The TAR Hairpin of Human Immunodeficiency Virus Type 1 Can Be Deleted When Not Required for Tat-Mediated Activation of Transcription

Stimulation of Tat-Associated Kinase-Independent Transcriptional Elongation From the Human Immunodeficiency Virus Type-1 Long Terminal Repeat by a Cellular Enhancer

Transcription Factor Binding Sites Downstream of the Human Immunodeficiency Virus Type 1 Transcription Start Site Are Important for Virus Infectivity.

Antiviral Activities of Glycyrrhizin and Its Modified Compounds Against Human Immunodeficiency Virus Type 1(hiv-1) and Herpes Simplex Virus Type 1(hsv-1) in Vitro.

Involvement of Long Terminal Repeat U3 Sequences Overlapping the Transcription Control Region in Human Immunodeficiency Virus Type 1 mRNA 3' End Formation.

Ribonucleotide Reductase of Herpes Simplex Virus Type 2 Resembles That of Herpes Simplex Virus Type 1.