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Publications by Wenlin Hao

Deficiency of Neuronal P38α MAPK Attenuates Amyloid Pathology in Alzheimer Disease Mouse and Cell Models Through Facilitating Lysosomal Degradation of BACE1

Journal of Biological Chemistry

English

Related publications

DNA Aptamers Targeting BACE1 Reduce Amyloid Levels and Rescue Neuronal Deficiency in Cultured Cells

Molecular Therapy - Nucleic Acids

Drug Discovery

Molecular Medicine

2019

English

Lack of BACE1 S-Palmitoylation Reduces Amyloid Burden and Mitigates Memory Deficits in Transgenic Mouse Models of Alzheimer’s Disease

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

2017

English

Myo-Inositol Changes Precede Amyloid Pathology and Relate toAPOEgenotype in Alzheimer Disease

Neurology

2016

English

Neuronal-Targeted TFEB Accelerates Lysosomal Degradation of APP, Reducing a Generation and Amyloid Plaque Pathogenesis

Journal of Neuroscience

Neuroscience

2015

English

Impact of RTN3 Deficiency on Expression of BACE1 and Amyloid Deposition

Journal of Neuroscience

Neuroscience

2014

English

P-Glycoprotein Deficiency at the Blood-Brain Barrier Increases Amyloid- Deposition in an Alzheimer Disease Mouse Model

Journal of Clinical Investigation

Medicine

2005

English

P38α-Mapk Signaling Inhibition Attenuates Soleus Atrophy During Early Stages of Muscle Unloading

International Journal of Molecular Sciences

Organic Chemistry

Molecular Biology

Theoretical Chemistry

Inorganic Chemistry

Computer Science Applications

English

Lysosomal NEU1 Deficiency Affects Amyloid Precursor Protein Levels and Amyloid-Β Secretion via Deregulated Lysosomal Exocytosis

Nature Communications

English

Review: Experimental Manipulations of Microglia in Mouse Models of Alzheimer's Pathology: Activation Reduces Amyloid but Hastens Tau Pathology

Neuropathology and Applied Neurobiology

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Wenlin Hao

Deficiency of Neuronal P38α MAPK Attenuates Amyloid Pathology in Alzheimer Disease Mouse and Cell Models Through Facilitating Lysosomal Degradation of BACE1

Related publications

DNA Aptamers Targeting BACE1 Reduce Amyloid Levels and Rescue Neuronal Deficiency in Cultured Cells

Lack of BACE1 S-Palmitoylation Reduces Amyloid Burden and Mitigates Memory Deficits in Transgenic Mouse Models of Alzheimer’s Disease

Myo-Inositol Changes Precede Amyloid Pathology and Relate toAPOEgenotype in Alzheimer Disease

Neuronal-Targeted TFEB Accelerates Lysosomal Degradation of APP, Reducing a Generation and Amyloid Plaque Pathogenesis

Impact of RTN3 Deficiency on Expression of BACE1 and Amyloid Deposition

P-Glycoprotein Deficiency at the Blood-Brain Barrier Increases Amyloid- Deposition in an Alzheimer Disease Mouse Model

P38α-Mapk Signaling Inhibition Attenuates Soleus Atrophy During Early Stages of Muscle Unloading

Lysosomal NEU1 Deficiency Affects Amyloid Precursor Protein Levels and Amyloid-Β Secretion via Deregulated Lysosomal Exocytosis

Review: Experimental Manipulations of Microglia in Mouse Models of Alzheimer's Pathology: Activation Reduces Amyloid but Hastens Tau Pathology